At the 12th hour, a slight elevation was detected in cardiac spesific troponins. We performed coroner angiography. There was not any luminal narrowing on coronary angiography but, 1.5-2 fold luminal dilatation which previously described coronary ectasy was detected on the 1/3 proximal portion of the left anterior descending coronary artery (LAD). Especially on the left lateral projection, opaque substance formed as an unique appearance of an inflated PTCA ballon and this continioued until the 120th frame (Figure 1 and Figure 2). Any lesion prodruding into the arterial lumen and coronary arterial dissection excluded by intravascular ultrasonography. Patogenesis of acute coronary synduome in our patient was unclear. But severe blood stegnation in the ectatic segment of LAD and thrombus formation and coronary slow flow due to microvascular atherosclerotic disease may explain the pathogenesis of the acute coronary syndrome.
Figure 1. Appearance of coronary ectasy in proximal of the Left Anterior Descending Coronary Artery.