Because of his history and existing symptoms, computed tomography (CT) angiography was performed, and circumferential narrowing was seen at the popliteal arteries; however, there was no thrombosis. The stenosis ratio was approximately 80% for the right and 60% for the left popliteal artery (Figures 1 and 2), and the bilateral popliteal artery was being compressed by the medial gastrocnemius tendons. The patient and his parents were informed of all of the findings and of the need for a reoperation. He was then referred to his cardiovascular surgeon.
Love and Whelan[5] developed most useful PAES scheme in 1965 and Rich et al.[6] were modified this scheme in 1979. According to this scheme PAES categorizied in to six subtypes:
Type 1: Medial head of gastrocnemius muscle is normal; popliteal artery is deviated medially and has an aberrant course.
Type 2: Medial head of gastrocnemius muscle is located laterally; no deviation of popliteal artery. Type 3: Abnormal muscle bundle from medial head of the gastrocnemius muscle surrounding the popliteal artery.
Type 4: Popliteal artery is located deeply and entrapped by the popliteus muscle or a fibrous band.
Type 5: Popliteal vein is also entrapped with any type of popliteal artery.
Type 6: Popliteal artery is normally positioned and entrapped by a normally positioned, hypertrophied gastrocnemius muscle.[3,6]
Color Doppler sonography is the primary imaging method used in the diagnosis of PAES, and it may also reveal popliteal artery stenosis, changes in color flow, or increased peak systolic velocity. Furthermore, this type of imaging also sometimes show the popliteal fossa anatomy and poststenotic aneurysms. Computed tomography angiography and magnetic resonance imaging (MRI) can show arterial stenosis, poststenotic dilatation, and the popliteal fossa anatomy. However, MRI is the best choice for evaluating the popliteal fossa anatomy and vascular compromise without the use of ionizing radiation or iodinated contrast material.[3,7]
Symptomatic PAES if not treated, eventually progresses the luminal narrowing, thrombosis and total occlusion. Because chronic microtrauma of the vessel wall can result in intimal damage and fibrosis that make the vessel susceptible to thrombosis.[8] The primary treatment method for PAES is surgery that involves extracting the muscle or tendons which causing the entrapment and also if necessary vessel lumen should be restored with endarterectomy or bypass grafting. The endovascular treatment of PAES not preferred choice without removing the underlying reason for the vessel entrapment however combined therapy-endovascular and surgical- had been performed.[8.9] Our patient had been operated two years before being admitted to our facility, and the popliteal arteries had been released via a myotomy and tenotomy. That surgery had alleviated the symptoms until three months prior to this admission when the symptoms had returned. A growing period or an inadequate release may have played a role in the recurrence of PAES in our patient.
In conclusion, PAES is usually seen in patients who are less than 30 years old and may also be found in teenagers, as in our case. Therefore, the pediatrician has to look for the presentation of claudication, which is the primary symptom of PAES. After surgery, teenage patients should be followed up closely to check for recurrence of this syndrome.
Declaration of conflicting interests
The authors declared no conflicts of interest with
respect to the authorship and/or publication of this article.
Funding
The authors received no financial support for the
research and/or authorship of this article.
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